having said that, offered the cross-sectional mother nature of these studies, causality for these associations cannot be decided. The adjustments in methylation may be a manifestation with the condition by itself. when these modifications may perhaps mirror important pathogenic mechanisms, the inference of causality assumed in these hypotheses is p
differing kinds of mutations (represented by in another way coloured arrows) happen at distinctive frequencies (represented by arrow thickness). Selection acts for a sieve and allows merely a subset of those mutations to persist and grow to be the discrepancies we see involving genomes. This is especially intriguing on condition that the now avail
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